CMI’s Joel Tay responds:
Thank you for writing to us.
Concerning the phrase, “Mutations cannot create new information”, it is necessary to read some of our more recent articles on this issue. In this article, “Can mutations create new information”, Dr Robert Carter explains why he does not agree with this statement.
There is a common myth that Human-Chimp DNA is 98% similar. This figure was based on some early experimental evidence (Reassociation kinetics) in 1975. What the researchers did was to extract DNA from two species, add them to the same test tube, warmed the tube up, and then measured how much light was absorbed by the combination as it cooled. These early reports were popularized by some evolutionists but this was long before even the initial drafts of the human and chimp genome that were announced in 2001 and 2005, respectively. As explained in Evolution’s Achilles’ Heels, and other places, with our modern understanding of genetics, we now know that “98%” is simply not the case.
Many portions of the genome are so different that a one-on-one comparison between human and chimpanzee DNA is impossible. For example, humans have several hundred protein-coding genes (all tightly integrated into the spliceosome) that are absent in chimps, the “similar” genes are scattered about in different places in the genome, and there are also entire gene families that are found in humans that are not in chimps. In the segments of the genome that are similar enough to make a comparison, the percentage of actual similarity between human and chimp DNA is probably closer to 80% – and when we compare the human and chimp Y chromosomes, the figure is even lower. These drastic differences between human and chimp DNA cannot be accounted for by evolutionary mathematics, so the statement that humans and chimps are 98% identical is nothing more than evolutionary dogma.
Evolutionists believe that humans and chimps split from a common ancestor 5–7 million years ago. As ‘evidence’, for decades, evolutionists have been claiming that humans and chimpanzees are nearly identical. One often hears “98% similarity”, or something similar. But is this true? Consider the following:
In the supposedly six million years since evolutionists believe humans and chimps split from a common ancestor, there is a need to account for 35,000,000 single letter differences that had to arise and become fixed in the two genomes (i.e. the original letter in that location was lost completely); tens of millions of chromosomal rearrangements had to occur, spread, and fix; as well as tens of millions of basepair insertions and deletions. (See Evolution’s Achilles’ Heels, p.75 for more details). In short, there is simply insufficient time for evolutionists to account for the differences between chimp and human DNA. Evolutionary time is measured in generations, not years. In six million years, there would only have been a few hundred thousand generations since chimps and humans were supposedly the same species. How then can there be enough time for so many brand-new genes to arise and be integrated? Each generation would have to select and retain an unbelievably huge number of mutations. This problem has come to be known as Haldane’s dilemma. Despite many claims to the contrary, Haldane’s Dilemma has never been solved. If anything, in recent years, our understanding of genetics has demonstrated that the problem is far greater for evolutionists than even Haldane imagined.1
Evolutionists have in the past cited junk DNA as a potential mechanism for solving this problem. ‘junk DNA’ refers to what was thought to be non-functioning portions of DNA. It was assumed that these were vestigial genomic ‘debris’ left behind from our evolutionary past. Since 97% of the DNA in the genome does not code for proteins, evolutionists proposed that these sections of ‘junk DNA’ would be free to mutate and evolve over time without significant consequences for the organism. The concept of junk DNA was extremely important for evolutionists as it was a last gasp in justifying how so many mutations could have arisen and remained fixed in the genome in such a short period of time (i.e. generations).
Unfortunately for the evolutionist, most scientists have now rejected the idea of junk DNA. The non-coding portions in the genome are now known to be almost completely functional. You might want to read these articles: here, here, and here on functions of ‘junk’ DNA. The general lack of junk DNA is yet another Achilles heel of evolutionary mathematics and argues strongly against biological evolution.
I would also highly recommend reading up on what Dr Carter calls the four dimensional genome. In my opinion, this is even more devastating to evolution than Haldane’s dilemma and the lack of Junk DNA. The four dimensional genome is one of my personal favorite argument against evolution from genetics. When the human genome was first sequenced, we thought that we could understand how the genome worked by sequencing the linear string of nucleotides. But this simplified understanding was naïve as we now know that this is only one of four dimensions of DNA as an information storage mechanism. If this is something you are interested in, I would highly recommend having a look at Dr Carter’s talk, The High Tech Cell, as well as Evolution’s Achilles’ Heels. Both resources discuss the four dimensional genome.
In a recent edition of Creation magazine (Vol. 38, No. 4, 2016), we also have an interview with Geneticist, Dr Jeffrey Tomkins, where he discusses human and chimp DNA. I have included an excerpt from that article that relates to your question. (See “Chromosome 2 Fusion?” below).
I hope that helps,
Humans have 23 chromosome pairs. Apes have 24. Evolutionists often claim that two smaller chromosomes fused to create human chromosome 2 at some early point in human history. They base this claim on the fact that the banding patterns in two smaller chimpanzee chromosomes are similar to the banding pattern on human chromosome 2. However:
Considering all this, there is little evidence that human chromosome 2 is the result of an ancient fusion event.